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8/12/2019 AKU KEGANASAN 191111.ppt
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Smiles for Life
1
KeganasanKeganasanRongga MulutRongga Mulut
goeno subagyo
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Speaking
Chewing
Tasting
Swallowing
Laughing
Smilling
Kissing
Happiness
Socializing
C.Everett Koop: You are not healthy without good oral health.
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Inte!Relationships "etween #al
an$ S%stemic Health an$ &isease
SIST'MI
K
#R(
L
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dokter gigi mempunyai tanggung jawab
profesional dan moral untuk mempelajarikesehatan umum pasien sebelum
memberikan perawatan dental
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Wanita usia 65 tahun dengan
polimedikasi untuk penyakit
kardiovaskuler, dengan rampant aries!karies yang "erkem"ang epat
meli"atkan "e"erapa gigi#
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Mani,estasi Keganasan $i Mulut
Signs S%mptoms
Keganasan
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Mani,estasi Keganasan $i Mulut
Sistemik
Keganasan
Lokal
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Mani,estasi #al keganasan
$on%&odgkin lymphoma
!large uler on the so't
palate#
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Mani,estasi #al Keganasan
Chroni lymphoyti leukemia
!uler on the palate#
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Mani,estasi #al Keganasan
Chroni lymphoyti leukemia!severe gingival enlargement and
ulerations#
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Mani,estasi #al Keganasan
(lkus Kronis )))))))))
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Mani,estasi #al Keganasan
Spontaneous gingial lee$ing o,
same patient with cihosis
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Mani,estasi #al Keganasan
Swollen shin% lee$ing gingia o,a patient with acute m%elogenous leukemia
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Smiles for Life
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Mani,estasi #al Keganasan
KE*+$++$
+$E-+
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Mani,estasi #al Keganasan
/aki%laki 60 tahun dengan
ulkus kronis pada sisi
lateral lidah kiri.
-empunyai ke"ia% saan
merokok dan minum
minuman keras.
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/aki%laki perokok 51th.
dg nonpain'ul lesion
pd dasar mulut
selama 1 "ln.
/eukoplakia
Candidiasis
/ihen
2lanus
3ral Caner
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#R(L C(4C'RSilent Kille
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#al Cance
5 #R#!6H(R748'(L C(4C'R
IS TH' 'L'9'4TH
M#ST C#MM#4 C(4C'R :#RL&:I&'
; :ol$ Health #ganization 200*
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#al Cance
30000 new cases $iagnose$ %eal%
.000 $eaths each %ea
* %ea suial ate< *0=
Oral cancer is the sixth most common cancer among
men
Inci$ence an$ Suial o, #al
o 6ha%ngeal Cance
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#al Cance
4 he overall survival rate 'or oral
aner in the general population is
50.
4 + survival rate 7orse than prostate,
uterine, "reast, "ladder and olonaners.
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#al Cance
5 ( * !%ea suial ate ,o (,ican (meican
men with oal cance is appo>imatel%
3)=?
5 :ith eal% $etection an$ teatment the *!
%ea %ea suial ate can incease up to
.*=?
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#al Cance
'al% &etection Saes Lies
5%year survival 'or loali8ed disease is95
5%year survival 'or metastati disease is;
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'al% Cance &etection
'al% #al Cance &etection is #,ten6ossile
issue hanges in the mouth that may
signal aner an o'ten "e seen and 'elt
easily
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Early detectionEarly detection ChemopreventionChemoprevention
High-risk:P53; ploidy
BDJ
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( 6ecanceous Lesion
( mophologicall% altee$ tissue in which
cance is moe likel% to occu than in itsappaentl% nomal countepat
@:H# pulication1//-AA
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Clinial /esions
+ssoiated 7ith 2remalignany
Leukoplakia'%thoplakiaLichen planus
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( pe$ominantl% white lesion o, the oalmucosa that cannot e chaacteize$ as an%
othe $e,inale lesion?
Leukoplakia
!W&3, ;;1#
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:hite Lesions
/ihen planus
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Leukoplakia< Clinical Beatues
5 Leukoplakia usuall% a,,ects people oe
the age o, )0 %eas @aeage age is +0
%easA?
5 6ealence inceases api$l% with age
paticulal% in males?
5 (ppo>imatel% . = o, the males oe the
age o, -0 %eas ae epote$l% a,,ecte$?
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Leukoplakia< Clinical Beatues
Continue$
5 (ppo>imatel% -0 = o, the oal
leukoplakias ae ,oun$ on the lip emilion
uccal mucosa an$ gingia?
5 4ote< Lesions o, the tongue lip emilion
an$ ,loo o, the mouth account ,o moe
than /0 = o, those that show $%splasia o
cacinoma upon histologic e>amination?
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Leukoplakia< Clinical Beatues
Continue$
5 In$ii$ual lesions a% in clinical appeaancean$ ten$ to change oe time?
5 'al%mil$ lesions usuall% appea as slightl%eleate$ ga% o ga%!white plaDues which ma%
appea tanslucent ,issue$ o winkle$ an$ aet%picall% so,t an$ ,lat?
5 'al%mil$ lesions ae usuall% well $emacate$ut ma% len$ into the suoun$ing nomal
mucosa?
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Leukoplakia< Clinical Beatues
Continue$
5 'al%mil$ thin leukoplakia which sel$om shows$%splasia on iops% ma% $isappea o continueunchange$?
5I, the cause @sA o, the lesion ae not emoe$man% lesions will ga$uall% ecome thicke an$lage?
5 The clinical appeaance @sA o, leukoplakia an$
the anticipate$ un$el%ing histopathologicchanges ae pesente$ in the ,ollowing $iagam?
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3)
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3*
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3+
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3-
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3.
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3/
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Homogeneous leukoplakia
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+ predominantly 7hite lesion 7ithverruous,nodular, ulerated or erythematous
'eatures.
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4#4!H#M#8'4'#ES L'EK#6L(KI(
4o$ula @4A an$ eucous @9A leukoplakia
49
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Leukoplakia
5 Tans,omation to $%splasia ates
! Thick leukoplakia F 1!-=
! 8anula leukoplakia F )!1*=
5 Tans,omation to SCC ates
! Seee $%splasia F 20!3*=
! Esuall% 2!) %eas a,te onset o, leukoplakia
! Risk incease$ i,persistent lesion, female patient,
nonsmoker, FOM or ventral tongue site
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Leukoplakia5 Teatmentamination
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Sceening '>amination
,o #al Cance
Sceening e>am eDuies