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“EFRUZHU CANCER (CARCİNOGENESİS)THEORY” DRUG THERAPY SUGGESTİO NS 26. =VO İCED 26. Capture_20120809_27.w m v 1. NORM AL DNA SEQ UENCE SUPPRESSİVE=REPRESSİVE AND İTS DNA GENES HYPERM ETHYLATED SO THAT TRANSCRİPTİON EXPRESSİON UNSUCCESSFUL AS DİFFERENT DEGEE DEPENDENCE OF ENERGY AND CELLSHOULD NOT COORDİNATE PHYSİOLOGİC M ETABOLİC ACTİVİTY. 1.1:W E NEED TO OPPENED HİSTONE PROTEİNS AND HYPOM ETHLATED CONDİTON TOGETHER REACTİVATİON AEROBİC GLYCOLYSİS,M İTHOCHONDRİON,HİGH ENERGY PHOSPHATE M OLECULESAND ANTİİNFLAM M ATİON +ANTİOXİDANCE APPLİCATİON. 2.M UTATİONS GROUP İDENTİTY THİS DNA SEQUENCE ORİGİN FROM OUTSİDE , GOVERN THE NORM AL CELL AS M ACHİNERY-DYNAM İCS DOM İNANTLY .M UTATİONAL DNA SEQ UENCE LİKE BAD DRİVER THAT M ANAGES ALLTHE

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“EFRUZHU CANCER (CARCİNOGENESİS) THEORY”

DRUG THERAPY SUGGESTİONS 26. =VOİCED 26.

Capture_20120809_27.wmv

1. NORMAL DNA SEQUENCE SUPPRESSİVE=REPRESSİVE AND İTS DNA GENES HYPERMETHYLATED SO THAT TRANSCRİPTİON EXPRESSİON UNSUCCESSFUL AS DİFFERENT DEGEE DEPENDENCE OF ENERGY AND CELL SHOULD NOT COORDİNATE PHYSİOLOGİC METABOLİC ACTİVİTY.

1.1:WE NEED TO OPPENED HİSTONE PROTEİNS AND HYPOMETHLATED CONDİTON TOGETHER REACTİVATİON AEROBİC GLYCOLYSİS,MİTHOCHONDRİON,HİGH ENERGY PHOSPHATE MOLECULES AND ANTİİNFLAMMATİON +ANTİOXİDANCE APPLİCATİON.

2.MUTATİONS GROUP İDENTİTY THİS DNA SEQUENCE ORİGİN FROM OUTSİDE , GOVERN THE NORMAL CELL AS MACHİNERY-DYNAMİCS DOMİNANTLY .MUTATİONAL DNA SEQUENCE LİKE BAD DRİVER THAT MANAGES ALL THE