Osteoarthritis Bt

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    OSTEOARTHRITIS

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    Osteoarthritis

    Primary generalizedfactors

    Secondary (followsdemonstrable abnormality

    & occurs following certainpredisposing factors) trauma

    congenital deformity

    inflamatory and infection

    metabolic disorder

    acquired incongruity of jointsurface

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    Inflammatory Vs Non-inflammatory around the joint

    Feature Inflammatory Noninflammatory

    Pain (when?)

    SwellingErythema

    Warmth

    Morning stiffness

    Systemic features

    ESR, CRP

    Synovial fluid WBC

    Examples

    Yes (AM)

    Soft tissueSometimes

    Sometimes

    Prominent

    Sometimes

    Frequent

    WBC >2000

    Septic, RA, SLE,

    Gout

    Yes (PM)

    BonyAbsent

    Absent

    Minor (< 30 )

    Absent

    Uncommon

    WBC < 2000

    OA, AVN

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    Articular Vs Periarticular

    Clinical feature Articular Periarticular

    Anatomic

    structure

    Painful site

    Pain on

    movementSwelling

    Synovium,

    cartilage,

    capsuleDiffuse, deep

    Active/passive,

    all planesCommon

    Tendon, bursa,

    ligament,

    muscle, boneFocal point

    Active, in few

    planesUncommon

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    Epidemiology

    Primary Type :

    females

    develops spontaneously in middle age

    progresses slowly

    Secondary Type :

    males

    develops at any ages as result of injury

    more rapidly progresses

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    Epidemiology

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    Pathogenesis Staging

    Initial stages

    Early stages

    Intermediate stage

    Terminal Stage

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    Pathology

    Cardinal features :

    1. Progressive cartdestruction

    2. Subarticular cystformation

    3. Sclerosis ofsurrounding bone

    4. Osteophytesformation

    5. Capsular fibrosis

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    Risk Factors

    Joint dysplasia Perthes disease

    Hurry prophylactic surgery in child

    Trauma # involving artic surf

    Malunion long-bone #

    repetitive stress in athletes

    Bone density

    Obesity

    Family history

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    Clinical Findings

    Pain Stiffness

    Swelling

    Deformity

    Loss of function

    Cartilage

    Deterioration

    Crepitus

    Osteophytes -Bone Spurs

    Loss of Joint

    Space Increased Density

    of Bone in Joint

    Lines Increased Synovial

    Fluid

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    12/4/2012

    Clinical Classification

    Monoarthritis Acute: Bacterial, Trauma, Crystal, Reactive Chronic :Tuberculosis, Lyme, Fungal, Trauma,

    Tumors

    Polyarthritis Autoimmune :Rheumatoid, Ankylosing spondylitis,

    Reiter syndrome Degenerative : Osteroarthritis CrystalDeposition :

    Gout Monosodium urateCPPD Pseudo Gout

    Infective Septic, TBC

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    Radiology Findings

    Narrowing of the

    joint space

    Sclerosis Osteophyte

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    Radiology Findings

    - joint space

    obliterated

    - sclerosis

    - osteophyte

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    Radiology Findings4 Cardinal signs :

    Narrowing of medial

    compartement

    Sclerosis of

    subchondral bone

    Osteophyte

    Subchondral cyst

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    Imaging

    X-rays 4 cardinal signs

    Radionuclide scanning

    99mTc-HDP

    activity in subchondralregions

    vasc & new bone formation

    Arthroscopy show cartilage damage

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    Management

    3 observation :

    symptoms characteristically wax & wane

    (pain) subside spontaneously for long periods

    some less painful w/ passage of time & no

    more than pain killer

    extreme, recognize by x-ray rapidly

    progressive type of OA reconstructivesurgery

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    Early Treatment

    Principles :

    1. Prevent and correction deformity

    2. Maintain movement & muscle strength3. Protect the joint from overload

    4. Relieve pain

    5. Modified daily activity

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    Early Treatment

    Education Weight Reduction

    wearing shockabsorbing shoes

    avoiding activitieslike climbing stairs

    Sport activity

    Diet Daily ActivityBiomechanics

    Pain Relief

    vascular

    decompression of

    subchondral bone redistribution of

    loading forces

    Physical therapy

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    Early Treatment

    Pharmaceuticals

    Acetaminophen

    NSAIDs

    COX-2 inhibitors

    Supplements

    Glucosamine

    Chondroitin sulfate

    Beta carotene

    Vitamins : C, D, E

    Hyaluronic Sodium

    HRT

    Estrogen

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    Physical Therapy Problemsof the Knee

    Weight bearing joints

    More symptomatically

    prevalent in knee

    Stiffness, swelling,pain

    Problems with:

    Walking Climbing stairs

    In and out of bathtubs

    and chairs

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    Physical Therapy Problems ofthe Hip

    Pain, stiffness, andsevere disability

    May feel symptoms inhip, groin, inner thigh,buttocks.

    Limits moving andbending.

    Problems with: Dressing

    Foot care

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    Physical Therapy Interventions

    Patient education on joint care Increase strength

    Increase aerobic capacity

    Warm water swimming

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    Physical Therapy Interventions

    Application of heat or

    cold packs

    Exercise and

    stretching Walking aids

    Support pillows

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    Total Joint Replacement

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    COMPLICATIONS

    Deformities

    Subluxation

    CapsularHerniation

    Loose bodies

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    Keropos Tulang

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    Apa itu Osteoporosis?

    Penipisan masa tulang

    sehingga tulang menjadi

    rapuh (fragil) dan mudah

    patah

    Suatu keadaan yang

    paling sering ditemukan

    pada usia tua

    Keadaan yang dapat

    dicegah

    http://www.exn.ca/news/images/1999/12/02/19991202-osteobig.jpg
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    Tulang Normal Osteoporosis

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    Pendahuluan

    Apakah sama dengan usia lanjut ?

    Dapat terjadi pada usia berapa saja :1. Bayi

    2. Anak-anak

    3. Dewasa

    4. Orang tua

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    Pendahuluan

    Orang tua :

    wanita >>> laki-laki

    Kenapaperbedaan hormone

    stok tulang yang ada

    aktifitaskegiatan sehari-hari

    ( activity of daily living)

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    Wanita

    Hormon: dihasilkan olehbeberapa kelenjar dalamtubuh dengan fungsi yang

    kompleks dan rumit Estrogen paling berperan :

    1. Jantung dan pembuluhdarah

    2. Fungsi organ wanita

    3.

    4. Tulang

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    Estrogen

    Menghambat fungsi sel penghancur tulang

    ( osteoclast) --utama

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    Tulang

    Selalu berubah :

    Dari dalam kandungan bayi anak

    anak dewasa muda tua

    Perubahan yang bersifat keseimbangan

    baik yang positif maupun yang negatif

    Jenis tulang

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    Jenis tulang

    Tulang kortikal

    TulangTrabekular

    B R d li C l

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    Bone Remodeling Cycle

    Bone lining cells

    Bone

    Osteoclasts

    Resorption by

    osteoclasts

    Osteoblasts

    Regulasi

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    RegulasiTulang

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    Progresifitas kehilangan masa tulang

    NormalOsteo-penia

    Osteo-porosis

    SevereOsteo-porosis

    TulangKortikal

    Tulangtrabekula

    Sincere thanks to Dr. Susan Ott forpermission to use the above images

    Standar Osteoporosis

    http://faculty.washington.edu/smotthttp://faculty.washington.edu/smotthttp://courses.washington.edu/bonephys/http://courses.washington.edu/bonephys/http://courses.washington.edu/bonephys/http://courses.washington.edu/bonephys/http://courses.washington.edu/bonephys/http://courses.washington.edu/bonephys/http://courses.washington.edu/bonephys/http://courses.washington.edu/bonephys/
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    Standar OsteoporosisWorld Health Organization (WHO)

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    Tulang

    Keseimbangan yang positif :tulang yang dibentuk >> yang

    dihancurkan tulang

    tumbuh

    Keseimbangan yang negatif :tulang yang dibentuk

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    Tulang

    Proses terbentuknya dan penghancuran

    tulang itu adalah alami

    Dipengaruhi oleh beberapa faktor yang

    diantaranya dapat dikontrol

    F kt ik b b O t i

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    Faktor resiko penyebab Osteoporosis

    Faktor yang tidak dapat dikontrol: Umur

    Jenis kelamin

    Status hormonal

    Ras

    Riwayat keluarga Postmenopause

    F kt d t dik t l

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    Faktor yang dapat dikontrol:

    Alkoholisme Minum kopi yang berlebihan

    Asupan kalsium & Vit D yang rendah

    Immobilisasi yang lama Kurang gerak

    Merokok

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    Perjalanan Osteoporosis

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    Masa tulang yang rendah + Kerusakaan

    struktur mikrotulang

    Tulang makin rapuh

    Peningkatan resiko

    patah tulang

    Penurunan kualitas

    tulang

    Penurunan kekuatantulang

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    Tulang

    Faktor yang mempengaruhi :

    1. Internal

    2. Eksternal

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    Tulang

    Faktor internal :

    1. Turunan ( gen)

    2. Penyakit bawaan ( gen)

    3. Ras ( bangsa)

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    Tulang

    Faktor eksternal :

    1. Persediaan tulang masa muda

    2. Aktifitas sehari-hari

    3. Obat-obatan

    4. Penyakit infeksi

    5. Penyakit kronis / metabolisme6. Merokok / kopi / alkohol

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    Keropos Tulang

    Penghancuran/ pembentukan tulang :

    alami

    Tidak perlu ditakuti.

    Yang penting mencegah komplikasi yaitu

    patah tulang

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    Keropos Tulang

    Faktor yang dapat dikontrol atau masih

    dapat dikontrol

    Pencegahan trauma atau benturan keras

    Obat-obatan ??

    Latihan yang sesuai

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    Keropos Tulang

    Penentuannya : diagnose

    penting untuk menentukan obat-obatan,

    latihan yang diperlukan dsb

    Jenis Osteoporosis ( Primer)

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    Jenis Osteoporosis ( Primer)

    Tipe I (Postmenopause osteoporosis)

    Karena kekurangan

    estrogen

    Lebih sering mengenai

    tulang spongiosa

    Umur 50- 70 thn

    Terutama pada wanita

    Patah tulang sering terjadi pada pergelangantangan dan tulang bealakang

    Type II (Osteoporosis

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    Type II (Osteoporosissenilis)

    Biasanya setelah umur 70tahun

    Wanita : Pria2 : 1

    Tipe III (Sekunder)Disebabkan penyakit

    tertentuWanita : Pria 1:1

    Gejala Klinis

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    Gejala Klinis

    Terselubung ("silent disease" )

    Nyeri punggung

    Tinggi berkurang

    Tambah bongkok

    Patah tulang akibat trauma ringan

    Akibat Osteoporosis

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    Akibat Osteoporosis

    Akib t O t i

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    Akibat Osteoporosis

    Kenapa terjadi patah tulang?

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    Kenapa terjadi patah tulang?

    Akibat Osteoporosis

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    Akibat Osteoporosis

    Akibat Osteoporosis

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    Akibat Osteoporosis

    Tempat yang sering terjadi patah tulang

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    p y g g j p g

    Deteksi Osteoporosis

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    Deteksi Osteoporosis

    DXA (Dual Energy X-ray Absorptiometry)

    Dasar Dasar Pencegahan

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    Osteoporosis

    C

    http://www.rockfordortho.com/osteoporosis/ostintro.htm
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    Calcium

    Sebagai pengobatandan pencegahanosteoporosis

    Vitamin D

    http://www.rockfordortho.com/osteoporosis/ostintro.htmhttp://www.rockfordortho.com/osteoporosis/ostintro.htmhttp://www.rockfordortho.com/osteoporosis/ostintro.htm
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    Vitamin D

    Vitamin D meningkatkan penyerapan kalsium

    Preparat Gabungan

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    Preparat Gabungan

    Exercise (Olah raga)

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    e c se (O a aga)

    Efek olah raga/ calcium

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    Efek olah raga/ calcium

    F ll P i (Hi d i j h)

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    Fall Prevention (Hindari jatuh)

    Resiko jatuh:

    Pengunaan obat

    penenang

    Gangguanpanglihatan

    Gangguan tungkai

    dan kaki

    Kelainan berjalan

    P i di h

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    Persiapan di rumah

    Kamar mandi bersihdan jangan licin

    Penerangan yang

    cukup Pegangan ditangga

    atau didinding

    Menjaga berat badan ideal

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    Menjaga berat badan ideal

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    Hindari

    merokok

    Senam Osteoporosis

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    Senam Osteoporosis

    Pengobatan

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    g

    Obat-obatan

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    Estrogens (Premarin, Ogen, Estrace, Estraderm,

    Estratab, dll)

    SERMs/selective estrogen receptor modulators(Raloxifene/Evista)

    Bisphosphonates (Alendronate/Fosamax andRisedronate/Actonel)

    Calcitonin/Miacalcin

    Konsultasi dengan dokter !!

    Pembedahan

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    Pembedahan

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